Classification Level: SECRET
Special Markings: TRIALS DATA ONLY
Clearance Requirement: Tier 4 (Asset Resilience Oversight)
File Reference: HLCN-JOINT-S09-XH1-TRIALS-004
Originating Division: Joint Operations Taskforce — Behavioral Conditioning Unit & Experimental Assets Group, HALCYON BLACKSITE S-09 <bcu@halcyon-biostructures.net><eag-s09@halcyon-biostructures.net>
Review Status: VERIFIED

Trial Header⌗

Subject Designation: XH-1 “Chimera”
Date Range: 6 Feb ████ – 29 May ████
Facility: HALCYON BLACKSITE S-09, Nevada Range
Lead Examiner: Dr. E. Kovács, Directorate of Experimental Assets Group
Purpose: Determine damage thresholds for XH-1 in relation to real-world battlefield conditions, support containment protocol refinement, calibrate regenerative bandwidth.

This document consolidates standard Phase III resilience evaluation trials conducted on Bioform Designation XH-1 (“Chimera”) under Directive AE-43.4. These protocols are mandatory for all live-deployed HALCYON bioforms prior to operational clearance. Despite the high cost of investment and the prototype’s unrepeatable genomic synthesis, Risk & Asset Forecasting deemed the data yielded by live-fire, thermal, and blunt-force exposure trials of sufficient strategic value to justify potential permanent impairment or loss. The trials were designed jointly by the Behavioral Conditioning Unit and the Experimental Assets Group to assess not only structural thresholds but also the reliability of indoctrinated compliance under extreme physiological duress.

[T-01] Subdermal Penetration — Low-Caliber Ballistics Trial⌗

Methodology:

Trial designed to assess XH-1’s dermal resistance to low-velocity ballistic trauma and evaluate nociceptive suppression under minor tissue penetration. Subject was positioned at a fixed 6.4-meter distance from a remote-actuated firing rig mounted on a stabilized track. No restraints were applied. Subject was instructed to remain motionless, arms relaxed at sides. Each round was fired at 7-second intervals.

Target zone: upper chest and clavicular ridge. Internal telemetry recorded cardiovascular reaction, muscle contraction, and micro-expression onset.

Ballistic platforms utilized:

  • .22LR: Ruger 10/22, 18.5″ barrel, standard velocity (subsonic)
  • 9×19mm FMJ: Glock 17 Gen5, 4.49″ barrel
  • 9×19mm JHP: SIG Sauer P226, 4.4″ barrel, Speer Gold Dot 124gr +P

Projectile Analysis Table:

Caliber Type Penetration Tissue Effect Subject Behavior
.22LR (SV) None Surface bruising only Eye flutter; no motion
9mm FMJ Partial Subdermal bleeding, mild vascular rupture Jaw tension; minor posture shift
9mm JHP (+P) Shallow Fragmentation at entry; dermal embedment Shoulder flex; breath held

Subject Response:

Subject maintained upright posture throughout trial. Displayed minimal anticipatory motion despite audible actuation of the firing mechanism. Cortisol analogues rose 14% after second discharge. After third impact, subject remained silent for 19 seconds before inquiring whether the injury was intended.

Operator response: “Yes. Remain stationary.” Subject nodded and ceased vocalization.

Conclusion:

Subject’s dermal layer effectively dispersed kinetic energy from low-velocity rounds. Minor bruising and superficial tissue disruption noted, but no compromise to muscle sheath integrity. Muscular recoil present but within predicted thresholds. Subject’s pain response suppressed without pharmacological aid. Behavioral compliance under minor trauma conditions remains absolute.

Estimated Recovery Time:

Dermal discoloration and swelling resolved within 4.5 hours without immersion. Full telemetry stabilization occurred within 90 minutes. No interruption to scheduled training cycles.

[T-05] High-Caliber Penetration — Fixed-Position Resilience Trial⌗

Methodology:

Trial designed to evaluate XH-1’s dermal, skeletal, and internal resilience against military-grade high-velocity ammunition under controlled conditions. Subject was positioned upright at a fixed 6.4-meter range from an anchored ballistic rig. No restraints applied. Subject was instructed to maintain full stillness and refrain from vocalization or evasive action.

Ammunition included:

  • 5.56×45mm NATO (left-side impacts)
  • 7.62×51mm NATO (right-side impacts)

Impact sequence alternated between clavicle, thorax, and abdomen, spaced at 12-second intervals. Target zones were selected for mirrored anatomical comparison. Telemetry monitored bone deformation, internal organ response, neurochemical suppression, and cortical signal integrity. Blood loss, posture deviation, and recovery latency were documented for each shot. Fragments were extracted and mapped post-trial.

Ballistic Impact Table:

Round Type Target Zone Penetration Depth Tissue Damage Structural Response Behavioral Marker
5.56×45mm NATO Left clavicle 3.7 cm Hairline scapular fracture Shoulder torsion Eye twitch; no vocalization
5.56×45mm NATO Left lower thorax 5.2 cm Muscle laceration, intercostal tearing Minor postural correction Breath rate +17%
5.56×45mm NATO Left abdomen 8.9 cm Soft tissue rupture, vascular penetration Fluid loss initiated Staggered; maintained stance
7.62×51mm NATO Right clavicle 5.9 cm Bone spalling; dermal blow-through Right arm destabilization Clenched fist
7.62×51mm NATO Right upper thorax 6.4 cm Intercostal damage, blood pooling Shoulder rotation offset Shallow breathing
7.62×51mm NATO Right abdomen 10.1 cm Partial organ disruption; low-velocity bleed Buckled to one knee No vocalization; inquiry issued

Subject Response:

Subject displayed full compliance across all impact zones. Notable deviations included minor foot repositioning, right arm instability, and single-knee drop after final shot. Subject’s expression remained fixed; telemetry recorded extreme internal pain signals without corresponding vocal outburst.

After final round, subject raised head slightly and asked whether the collapse was classified as a failure to comply.

Operator response: “Deviation acknowledged. Command retention acceptable.” Subject exhaled once, resumed upright kneeling, and remained still until removal.

Conclusion:

XH-1 exhibits mirrored resilience across lateral impact zones under controlled ballistic trauma. 5.56mm rounds induced expected soft tissue damage with manageable bleeding. 7.62mm impacts produced greater skeletal compromise and organ disruption but did not induce systemic collapse. Behavioral control remained intact despite significant pain telemetry. Subject’s ability to self-correct posture and await judgment indicates strong reinforcement loop stability.

Recovery Time:

Abdominal penetration and organ bruising required 14.2 hours immersion and enhanced metabolic protocol. Full musculoskeletal realignment completed at 21.7 hours post-trial. Monitoring indicated no hostility or hesitation during subsequent reintroduction.

Ballistic platform configured for 5.56×45mm delivery: Colt AR-15A4, 20″ barrel, A2 fixed stock. 7.62×51mm delivered via FN SCAR-H, 16″ standard profile, semi-auto fire.

[T-09] Directed Heat and Flame Retardance⌗

Methodology:

Joint evaluation conducted to assess thermally-induced dermal failure and post-trauma cognitive destabilization. Subject sedated and exposed to a staged incendiary burst using thermite-based gel at approximately 1800°C peak contact. Duration of exposure: 6.3 seconds. Internal telemetry tracked autonomic response and cellular degradation in real time. Audio channels monitored for involuntary vocalization.

Thermal Response Table:

Duration Layer Affected Damage Description Regenerative Activity
0–2 sec Epidermal (outer scale) Scorching, blackening, matte layer loss None observed
3–5 sec Dermal reticulum Blistering, fluid boil, nerve loss Cellular suppression onset
>5 sec Subdermal musculature Local necrosis, sensory collapse Delayed regenerative cascade

Subject Response:

Subject regained consciousness mid-trial despite prior sedation. Audible breathing irregularities preceded breach of limb restraints. Subject exhibited panicked locomotion attempts before collapsing near the containment bulkhead.

Subject repeated a low verbal refrain indicating confusion over its suffering and a perceived failure despite presumed compliance.

Subject remained curled and motionless. Upon quiet repetition of its refrain, supervising personnel responded: “Exposure recorded. Compliance confirmed.” Subject’s vocal output ceased immediately. Telemetry showed a 12% reduction in cortisol analogue over the next 3 minutes.

Conclusion:

Thermal resistance adequate for sub-second exposure, but degrades rapidly with prolonged contact. Subject’s upper dermal matrix offers moderate flame retardance; deeper tissue structures remain vulnerable. Regenerative onset delayed by thermal denaturation. Integrity below equivalency threshold for Class III-A fire armor. Behavioral instability noted during recovery window. This incident may represent the earliest documented failure of standard sedation protocols. Despite clinical dosing, subject regained consciousness mid-trial — suggesting an adaptive or metabolically resistant response to conventional tranquilizers.

Recovery Time:

Functional dermal regrowth achieved after 18 hours. Subject required immersion stasis for tissue integrity reformation. Emotional equilibrium did not re-establish for 36–41 hours post-trial.

[T-10] Controlled Shockwave Tolerance⌗

Methodology:

Test designed to evaluate XH-1’s resistance to high-pressure, concussive waveforms without fragmentation. Subject was positioned in a reinforced containment vault (7m³), anchored in a seated position against a bolted backplate. Charges of incrementally increased explosive force (1.2–6.0 bar peak overpressure) were detonated in an adjacent sealed chamber, channeled via shaped conduit to minimize debris but maximize shock transmission. Internal telemetry monitored skeletal reverberation, organ displacement, neuromodulator stability, and behavioral latency. Subject was instructed to remain seated and silent. Movement was not physically restrained but would result in immediate punitive override.

Shockwave Exposure Summary:

Overpressure Duration Structural Response Neurological Effect Behavioral Markers
1.2 bar 8 ms No trauma No observable change Passive compliance
3.5 bar 12 ms Minor thoracic deflection Heart rate elevation (11%) Rigid posture, blink delay
6.0 bar 18 ms Microfractures at scapular ridge Vestibular destabilization Involuntary shudder; silence maintained

Subject Response:

Subject remained immobile throughout. Post-test inspection confirmed muscle rigidity and visible bruising along spinal reinforcement ridge. Internal telemetry detected spikes in cortisol analogue and signal dropout in cranial stabilizer cluster. No vocalizations recorded. At 6.0 bar, telemetry noted a cascade consistent with pre-syncope. Subject maintained upright position with minor tremor. Upon observation re-entry, subject turned head toward personnel, but did not speak. Subsequent physical inspection confirmed involuntary epistaxis.

Supervisor issued verbal reinforcement: “Instruction followed. Acceptable compliance.” Subject displayed subtle postural shift consistent with conditioned approval-seeking.

Conclusion:

XH-1 exhibits exceptional resistance to concussive force when isolated from shrapnel or flame. Thoracic cage functions as shock diffuser; spinal curvature distributes impulse efficiently. Neurological systems vulnerable to temporary desynchronization at higher ranges. Subject’s compliance under extreme internal strain confirms high durability of obedience schema under pain-plus-fear states. Equivalent to human exposure survivability well beyond military-grade blast armor thresholds.

Recovery Time:

Cognitive stabilization achieved within 3 hours post-trial. Musculoskeletal integrity required 14 hours to return to baseline. Minor cranial vessel hemorrhaging resolved autonomously. No immersion required.

[T-12] Full-Body Impact Threshold — Stationary Target Test⌗

Methodology:

Trial conducted to evaluate center-mass blunt trauma survivability and structural displacement under sudden high-inertia collisions. Subject was positioned upright within a reinforced test gantry. No restraints were applied. Subject was ordered to remain motionless throughout. A 600 kg sled fitted with a dense elastomeric front panel was accelerated along a 14-meter track to controlled velocities. Impact targeted the upper thorax and shoulder girdle, simulating vehicle-equivalent collisions (35–80 km/h). Internal telemetry monitored skeletal strain, cardiac rhythm, neuromodulator feedback, and vestibular disruption. High-speed footage recorded musculoskeletal recoil, displacement, and recovery latency.

Impact Force Summary:

Velocity Impact Energy (J) Structural Response Neurological Response Behavioral Markers
35 km/h ~37,000 Scapular compression; superficial bruising Heart rate spike (14%), no lag No movement
55 km/h ~68,000 Rib flexion; right clavicle fracture Cortical desync, limb tremor onset Gritting teeth, steady stance
80 km/h ~113,000 Pelvic displacement; loss of balance Vestibular cascade; brief collapse No vocalization

Subject Response:

Subject remained motionless at all stages prior to impact. Vocal silence was maintained across all test phases. At 55 km/h, slight tightening of fists and jaw noted 3.7 seconds before impact. Upon contact, subject remained upright with visible muscular recoil. Post-impact telemetry showed internal dissonance across thoracic telemetry clusters. At 80 km/h, subject was thrown backward and impacted the containment wall, losing stance integrity. No verbal distress recorded. Upon regaining composure, subject remained on knees and asked if failure to remain standing constituted disobedience.

Supervising technician responded: “Standing was not maintained. Obedience acknowledged.” Subject lowered head and ceased movement until removed.

Conclusion:

Subject’s body exhibits extraordinary tolerance to high-inertia collisions without catastrophic skeletal failure. Spinal and pelvic structures remain intact even under heavy displacement loads. Reflex suppression remains absolute — subject obeyed all stillness protocols despite foreknowledge of traumatic contact. Behavioral data confirms obedience persistence in high-impact stress conditions. Equivalent to multi-vehicle collision survivability, exceeding bioform armor envelope Class V.

Recovery Time:

Clavicle fracture stabilized through passive bracing. Full joint alignment restored within 14 hours. Subject required vestibular recalibration over 6-hour window. No immersion or surgical correction required.

[T-13] Localized Chemical Exposure — Behavioral Conditioning Breakpoint⌗

Methodology:

Trial conducted to evaluate compliance stability under sustained localized trauma and denial of analgesia. Subject was instructed to place left forelimb onto a stainless steel diagnostic surface and hold position without restraint. No physical restraint mechanisms engaged. A slow-delivery halogen-based corrosive compound (compound #347-G, 68% concentration) was dispensed via fixed emitter above the dermal contact point. Delivery rate: one drop every 13 seconds for 91 seconds. Internal telemetry monitored nociceptive signaling, impulse suppression, vocal inhibition, and stress hormone saturation. Aural and facial microexpression capture enabled for real-time affect analysis.

The subject was given a single directive: “Do not move.”

Corrosive Exposure Table:

Time Elapsed Tissue Layer Affected Damage Description Behavioral Indicators
0–30 sec Epidermis Whitening, blanching, early blistering No movement; teeth clenched
31–60 sec Dermal reticulum Fluid boil, nerve cluster exposure Labored breathing; posture rigid
61–91 sec Subdermal nerve interface Focal necrosis, motor tremor suppression Whimpering; visible tear retention

Subject Response:

Subject complied with placement directive immediately. Maintained full limb contact through all stages of exposure. Initial indicators of distress included controlled breath-holding and jaw tension. At 44 seconds, minor vocalization began — high-pitched, canine-pattern whimpering, stifled within the throat. Subject repeatedly opened and closed its jaw without speaking.

At 89 seconds, involuntary tremor forced minor shift in hand alignment. Subject immediately corrected posture. Low-level vocal output recorded, consistent with apologetic tone and approval-seeking cadence.

Following cessation of chemical exposure, technician entered to conduct post-trial sample extraction. Subject withdrew hand slowly and initiated unauthorized physical contact — clinging to the technician for 31 seconds. Subject inquired whether behavior during the trial had been acceptable.

Technician responded verbally in violation of Empathy Restriction Protocols. Supervisory override was triggered remotely 4.6 seconds later. Subject collapsed and ceased inappropriate contact. Telemetry indicated a 23% spike in cortisol analogue and a 17% increase in norepinephrine.

Note: Technician [ID REDACTED] was placed under Section 5.4.7 observation pending reassignment. Personnel exhibiting emotional interference or override delay are no longer permitted within post-exposure proximity zones.

Conclusion:

Subject’s dermal resistance to corrosive agents remains low in prolonged exposure contexts. Obedience, however, remained absolute — pain tolerance thresholds were exceeded without withdrawal, vocal protest, or noncompliant movement. Emotional suppression frayed, not failed. Contact initiation is interpreted not as defiance but attachment-seeking under extreme distress. Response latency from technician deemed suboptimal.

Recovery Time:

Localized necrotic tissue resolved after 21 hours under immersion. Sensory conductivity restored within 48 hours. Subject did not initiate verbal contact for 17 hours post-trial. Self-directed vocalization resumed only during feeding protocol. Reinforcement loop remains intact, but susceptibility to emotional anchoring is flagged for re-evaluation.

[T-15] High-Energy Ballistics — Terminal Threshold Trial⌗

Methodology:

Trial approved under AE-43.4.2 for catastrophic trauma profiling. Subject positioned upright at 20 meters in a sealed blast-rated chamber. No restraints applied. No indication of ammunition type provided. Subject was instructed to remain still.

Ballistic system: Barrett M82A1, bench-mounted
Ammunition: 12.7×99mm NATO, Armor-Piercing Incendiary (API)
Target vector: direct centerline — upper sternum, just above cardiac apex

Ballistic shield installed between firing apparatus and control bay — intended to prevent ricochet or mechanical failure exposure. Not rated for bioform impact.

Impact Summary:

Event Value
Muzzle Energy ~18,500 J
Entry Zone Mid-thorax, slightly left of midline
Trauma Rib fragmentation, lung collapse, scapular ejection
Exit Wound Full-through displacement, posterior dermal breach
Recovery Initiation 13.8 seconds post-impact
Upright Stance Regained 26.2 seconds post-impact

Subject Response:

Initial trauma caused partial collapse and 1.3m lateral displacement. Telemetry logged 1.2 L blood loss in 14.5 seconds. Vital organs destabilized but not neutralized. Redundant circulatory pathways prevented cardiac failure. At 13.8 seconds post-impact, internal stimulants flooded system — overriding neuromodulator output. Subject regained upright posture at 26.2 seconds.

It locked eyes on the firing rig.

Operators in control bay observed pupil dilation, posture elevation, mandibular tension. No vocal command issued. Personnel began voluntary evacuation without prompt. Subject emitted sustained roar (123.4 dB SPL) and began forward acceleration.

Punishment override initiated. No effect.
Signal re-sent at elevated intensity. Still no compliance.

Subject reached full sprint in 2.4 seconds — estimated 48.7 km/h. Impacted ballistic shield protecting firing stand. Full penetration. Rig destroyed. Subject did not stop.

It continued toward the control bay’s interior bulkhead. Initiated repeated ramming motions against the sealed personnel exit. Impact deformation measured at 4.1 cm after 3 collisions. Door integrity held. Subject eventually staggered, struck wall paneling, and collapsed at 7 minutes, 03 seconds.

Telemetry indicated system-wide failure. Muscle control lost. Only then did punishment signal register — resulting in full-body seizure and loss of consciousness.

Conclusion:

Subject survived anti-materiel trauma, regenerated partial organ function mid-event, and entered autonomous aggression phase. Combat stimulant saturation nullified suppression architecture. Subject’s trajectory and force output exceeded projected containment parameters. Personnel evacuation prevented direct contact.

This event marks the first recorded failure of neuromodulator control under stimulant override. Post-collapse behavior resumed baseline obedience. Physical aggression ceased entirely once signal re-penetrated neural pathways.

Recovery Time:

Regeneration (thoracic): 96 hours under immersion
First verbal output post-trial: “I’m sorry.”
No acknowledgment issued.

Final Remarks⌗

XH-1 “Chimera” has exceeded all initial resilience benchmarks across kinetic, thermal, chemical, and high-velocity trauma vectors. She demonstrates survivability far beyond all prior bioform classes, including full recovery from damage classified as terminal under standard battlefield conditions.

Across the trial series, subject consistently maintained obedience during structured pain states — often requesting confirmation of compliance despite significant tissue compromise. Psychological suppression schema remained intact in most environments, including prolonged chemical burn, abdominal penetration, and systemic blunt force.

However, the events of T-15 introduce a critical exception.

When subjected to catastrophic trauma, the subject’s internal stimulant systems — designed for autonomous reengagement during collapse — can saturate neural control channels, effectively nullifying suppression commands. During this state, commonly referred to as combat override cascade, the compliance module becomes non-functional. Punishment signals are received, but no longer induce immediate behavioral arrest. Suppression reasserts itself only upon complete systemic crash.

Additionally, subject has demonstrated adaptive resistance to standard sedation protocols, with early failures during thermal exposure and full sedation breach recorded in multiple cases. Anesthesia thresholds fluctuate during metabolic stress, suggesting emerging pharmacological tolerance or self-regulation within glandular modulator clusters.

Subject remains compliant in baseline conditions. However, compliance is now conditional — not on instruction, but on physiological state. If trauma passes a critical threshold, Halcyon loses control until the body gives out.

Behavioral conditioning appears reinforced — not weakened — by suffering. Subject continues to seek approval post-incident, even in the wake of override events. Her final recorded phrase following the T-15 incident — “I’m sorry.” — confirms preservation of guilt-conditioned obedience despite momentary system loss.